Search results for "Malignant cells"

showing 5 items of 5 documents

Selective Cytotoxic Activity of Prodigiosin@halloysite Nanoformulation

2020

Prodigiosin, a bioactive secondary metabolite produced by Serratia marcescens, is an effective proapoptotic agent against various cancer cell lines, with little or no toxicity toward normal cells. The hydrophobicity of prodigiosin limits its use for medical and biotechnological applications, these limitations, however, can be overcome by using nanoscale drug carriers, resulting in promising formulations for target delivery systems with great potential for anticancer therapy. Here we report on prodigiosin-loaded halloysite-based nanoformulation and its effects on viability of malignant and non-malignant cells. We have found that prodigiosin-loaded halloysite nanotubes inhibit human epithelia…

0301 basic medicineHistologylcsh:BiotechnologyBiomedical EngineeringBioengineering02 engineering and technologyhalloysite nanotubesengineering.materialHalloysiteProdigiosin03 medical and health scienceschemistry.chemical_compoundcomet assaylcsh:TP248.13-248.65Cytotoxic T cellcancerOriginal Researchgenotoxic effectanti-cancer drugsbiologyChemistryBioengineering and Biotechnology021001 nanoscience & nanotechnologybiology.organism_classificationmalignant cellsComet assay030104 developmental biologyprodigiosinDrug deliveryToxicitySerratia marcescensdrug deliveryCancer researchengineering0210 nano-technologyDrug carrierBiotechnologyFrontiers in Bioengineering and Biotechnology
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Coupling tumor necrosis factor‐related apoptosis‐inducing ligand to iron oxide nanoparticles increases its apoptotic activity on HCT116 and HepG2 mal…

2019

International audience; Tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) has been considered as a potential anticancer agent owing to its selectivity for malignant cells. However, its clinical use remains limited because of its poor efficacy. Attempts to increase its antitumor activity include, among others, its functionalization by nanoparticles (NPs). In the present study, TRAIL was grafted onto magnetic spinel iron oxide NPs of defined core size, 10 and 100 nm on average, to see whether the size of the resulting nanovectors, NV10 and NV100, respectively, might affect TRAIL efficacy and selectivity. Apoptosis induced by NV10 and NV100 was higher than by TRAIL alone in both …

0303 health sciencesLigand (biochemistry)3. Good healthCoupling (electronics)03 medical and health scienceschemistry.chemical_compound0302 clinical medicinechemistryMagnetic coreApoptosis030220 oncology & carcinogenesis[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular BiologyBiophysicsMalignant cellsTumor necrosis factor alpha[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyIron oxide nanoparticles030304 developmental biology
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Does Exist A Correlation Between BMI and Gleason Patterns 4 and 5 At Prostate Biopsy?

2013

Background: Obesity has been related with higher Gleason grade and worse prognosis. Increasing proportion of Gleason pattern 4 or 5 is a critical factor for biochemical recurrence, progression and mortality. The endocrine activity of visceral fat could be responsible of the differentation of the prostatic malignant cell towards a more aggressive fenotype. The aim of our study was to correlate Body Mass Index with the presence of Gleason pattern 4 or higher at biopsy. Materials and Methods: Consecutive patients with positive prostate biopsy were included. A transrectal prostate biopsy procedure with 12 cores, was performed. All tissue samples were reviewed. Results: Out of 135 patients diagn…

Gleason gradeOncologyBiochemical recurrencemedicine.medical_specialtyProstate cancerProstate biopsymedicine.diagnostic_testbusiness.industryUrologyurologic and male genital diseasesGleason grademedicine.diseaseSettore MED/24 - UrologiaCorrelationBMIProstate cancerProstate BiopsyInternal medicineBiopsymedicineMalignant cellsObesitybusinessBody mass indexJournal of Cytology & Histology
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Estrogen-Induced Mutations and Its Role in the Development of Tumorigenesis

2006

Using RAPD-PCR fingerprinting, the detection of mutations induced by estrogen (E) exposure was studied in nonmalignant- and malignant cells. Cells exposed to 17α-and 17β-estradiol, diethylstilbestrol, bisphenol A, and α-zearalanol induced mutations in different regions of the genome. They consisted of insertion/deletion as a result of point or length mutations and quantitative changes as a result of hypoploidy or polyploidy. We also detected several mutated loci in tumor-free tissues, adjacent to tumors, and DES-induced tumors. The data suggest that these mutations occurred in early stages of stilbene E-induced renal carcinogenesis, and that they may be induced as a result of stilbene E tre…

HypoploidyEstrogenmedicine.drug_classCancer researchDiethylstilbestrolmedicineMalignant cellsBiologyCarcinogenesismedicine.disease_causeRenal carcinogenesisGenomemedicine.drug
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Novel therapeutic targets in esophageal cancer: impact of chemokine receptor CXCR4

2007

Ines Gockel†, Carl C Schimanski, Markus Moehler & Theodor Junginger †Author for correspondence Johannes GutenbergUniversity of Mainz, Department of General and Abdominal Surgery, Langenbeckstr. 1, 55131 Mainz, Germany Tel.: +49 6131 177 291; Fax: +49 6131 176 630; gockel@ach.klinik.unimainz.de ‘The interaction between esophageal cancer-expressed CXCR4 and SDF-1α may have a key role in directing malignant cells to ‘homing’ organs ... thus, this mechanism may account for metastasis.’

Receptors CXCR4Cancer ResearchEsophageal Neoplasmsbusiness.industryAntineoplastic AgentsGeneral MedicineEsophageal cancermedicine.diseaseCXCR4Chemokine CXCL12Cyclin-Dependent KinasesNeoadjuvant TherapyhumanitiesChemokine receptorDrug Delivery SystemsOncologyCancer researchmedicineHumansMalignant cellsbusinessChemokines CXCHoming (hematopoietic)Future Oncology
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